6801 to 6850 MCQs for Lab Technician and Technologist Exam Preparation
5000 Plus MCQs for Lab Technician and Technologists are designed to test the knowledge and proficiency of laboratory professionals who work in the field of clinical laboratory science. These questions cover a wide range of topics related to laboratory science, including anatomy, physiology, microbiology, chemistry, and hematology.
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Questions 6801 to 6850
- Ceramide is formed by the combination of sphingosine and
- Acetyl-CoA
- Acyl-CoA ✔
- Malonyl-CoA
- Propionyl-CoA
- The amino alcohol sphingosine is synthesized in
- Mitochondria
- Cytosol
- Nucleus
- Endoplasmic reticulum ✔
- The output of free fatty acids from adipose tissue is reduced by
- Insulin ✔
- Glucagon
- Growth hormone
- Epinephrine
- The principal action of insulin in adipose tissue is to inhibit the activity of the
- Hormone sensitive lipoprotein lipase ✔
- Glycerol phosphate acyltransferase
- Acetyl-CoA carboxylase
- Pyruvate dehydrogenase
- In non shivering thermogenesis
- Glucose is oxidized to lactate
- Fatty acids uncouple oxidative phosphorylation ✔
- Ethanol is formed
- ATP is burned for heat production
- Brown adipose tissue is
- A prominent tissue in human
- Characterised by high content of mitochondria ✔
- Associated with high activity of ATP synthase
- Characterised by low content of cytochromes
- Fatty liver is caused due to accumulation of
- Fatty acids
- Cholesterol
- Phospholipids
- Triacylglycerol ✔
- A lipotropic factor is
- Choline ✔
- Palmitic acid
- Calcium
- Vitamin C
- Fatty liver is also caused by
- CH3Cl
- CCl4 ✔
- Na2SO4
- Riboflavin
- All the enzymes involved in the synthesis of cholesterol are found in
- Mitochondria
- Golgi apparatus
- Nucleus
- Endoplasmic reticulum and cytoso ✔
- The source of all the carbon atoms in cholesterol is
- Acetyl-CoA ✔
- Bicarbonate
- Propionyl-CoA
- Succinyl-CoA
- Two molecules of acetyl-CoA condense to form acetoacetyl-CoA catalysed by
- Thiolase ✔
- Kinase
- Reductase
- Isomerase
- Acetoacetyl-CoA condenses with one more molecule of acetyl-CoA to form
- Mevalonate
- Acetoacetate
- β-Hydroxybutyrate
- 3-Hydroxy 3-methyl-glutaryl-CoA✔
- HMG-CoA is converted to mevalonate by reduction catalysed by
- HMG-CoA synthetase
- HMG-CoA reductase ✔
- Mevalonate kinase
- Thiolase
- For reduction enzyme HMG-CoA reductase requires cofactor:
- NADPH ✔
- NADP
- NAD
- FAD
- In the biosynthesis of cholesterol, the step which controls the rate and locus of metabolic regulation is
- Geranyl pyrophosphate farnesyl pyrophosphate
- Squalene → lanosterol
- HMG CoA → mevalonate ✔
- Lanosterol → 1, 4-desmethyl lanosterol
- The cyclisation of squalene in mammals results in the direct formation of the sterol.
- Cholesterol
- Lanosterol ✔
- Sistosterol
- Zymostero
- In the biosynthesis of cholesterol, the rate limiting enzyme is
- Mevalonate kinase
- HMG-CoA synthetase
- HMG-CoA reductase ✔
- Cis-prenyl transferase
- Cholesterol by a feed back mechanism inhibits the activity of
- HMG-CoA synthetase
- HMG-CoA reductase ✔
- Thilase
- Mevalonate kinase
- The activity of HMG-CoA reductase is inhibited by
- A fungal inhibitor mevastatin ✔
- Probucol
- Nicotinic acid
- Clofibrate
- Hypolipidemic drugs reduce serum cholesterol and triacylglycerol. The effect of clofibrate is attributed to
- Block in absorption from G.I.T.
- Decrease in secretion of triacylglycerol and cholesterol containing VLDL by liver ✔
- Block in the reabsorption of bile acids
- Decreased synthesis of cholesterol
- In biosynthesis of cholesterol triparanol inhibits the activity of the enzyme:
- ∆24 Reductase ✔
- Oxidosqualene-lanosterol cyclase
- Isomerase
- Squalene epoxidase
- HMG-CoA reductase activity is increased by administration of the hormone:
- Insulin ✔
- Glucagon
- Epinephrine
- Glucocorticoids
- The principal sterol excreted in feces is
- Coprostanol ✔
- Zymosterol
- Lanosterol
- Desmosterol
- The principal rate limiting step in the biosynthesis of bile acids is at the
- 7-Hydroxylase reaction ✔
- 12 α-Hydroxylase reaction
- Conjugation reaction
- Deconjugation reaction
- Hypercholesterolemia is found in
- Xanthomatosis ✔
- Thyrotoxicosis
- Hemolytic jaundice
- Malabsorption syndrom
- Hypocholesterolemia is found in
- Thyrotoxicosis ✔
- Diabetes mellitus
- Obstructive jaundice
- Nephrotic syndrome
- The major source of extracellular cholesterol for human tissue is
- Very low density lipoprotein
- High density lipoprotein
- Low density lipoprotein✔
- Albumin
- Correct ordering of lipoprotein molecules from lowest to the greater density is
- LDL, IDL, VLDL, chylomicron
- Chylomicron, VLDL, IDL, LDL ✔
- VLDL, IDL, LDL, chylomicron
- LDL, VLDL, IDL, chylomicron
- In Hurler’s syndrome, urine shows the presence of
- Keratan sulphate I
- Chondroitin sulphate
- Dermatan sulphate and heparan sulphate ✔
- Keratan sulphate II
- Defective enzyme in Hunter’s syndrome is
- α-L-iduronidase
- Iduronate sulphatase✔
- Arylsulphatase B
- C-acetyl transferase
- An important feature of Von-Gierke’s disease is
- Muscle cramps
- Cardiac failure
- Hypoglycemia ✔
- Respiratory alkalosis
- The affected organ in Mc Ardle’s syndrome is
- Liver
- Kidney
- Liver and Heart
- Skeletal muscle✔
- Refsum’s disease is due to deficiency of the enzyme:
- Pytantate-α-oxidase ✔
- Glucocerebrosidase
- Galactocerebrosidase
- Ceramide trihexosidase
- An important finding in Refsum’s disease is
- Accumulation of ceramide trihexoside in the kidney
- Accumulation of phytanic acid in the blood and tissues ✔
- Accumulation of gangliosides in brain and spleen
- Skin eruptions
- α-Galactosidase enzyme is defective in
- Tay-sach’s disease
- Refsum’s disease
- Sandhoff’s disease
- Fabry’s disease ✔
- The hypothesis to explain enzyme– substrate complex formation:
- Lock and key model
- Induced fit theory
- Proenzyme theory
- Both (Lock and key model) and (Induced fit theory) ✔
- An important finding in Tay-sach’s disease is
- Renal failure
- Accumulation of gangliosides in brain and spleen ✔
- Cardiac failure
- Anemia
- The enzyme deficient in Krabbe’s disease is
- Hexosaminidase A
- Arylsuphatase A
- β-Galactosidase✔
- α-Fucosidase
- The enzyme ceramidase is deficient in
- Farber’s disease ✔
- Fabry’s disease
- Sandhoff’s disease
- Refsum’s disease
- Niemann-Pick disease is due to deficiency of the enzyme
- Ceramidase
- Glucocerebrosidase
- Galactocerebrosidase
- Sphingomyelinase ✔
- The enzyme deficient in Sandhoff’s disease is
- α-Fucosidase
- Hexosaminidase A and B ✔
- β-Galactosidase
- β-Glucosidase
- Jamaican vomiting sickness is due to inactivation of the enzyme
- Pyruvate carboxylase
- Acyl-Co-A synthetase
- Acyl-Co-A dehydrogense ✔
- Thiolase
- Bassen-Kornzweig syndrome is due to
- Absence of Apo-C-II
- Defect in Apo-B synthesis ✔
- Absence of Apo-E
- Absence of Apo-D
- Enzyme deficient in Hyperammonemia type II is
- Glutamine synthetase
- Glutaminase
- Ornithine transcarbamoylase ✔
- Carbamoylphosphate synthetase
- An important finding in Hyperammonemia type II is
- Increased serum gluatmine level
- Enlarged liver
- Mental retardation
- Increased carbamoyl phosphate synthetase level ✔
- Absence of the enzyme argininosuccinate synthetase causes
- Argininosuccinic aciduria
- Hyperargininemia
- Tricorrhexis nodosa
- Citrullinemia ✔
- Tricorrhexis nodosa is a characteristic finding of
- Argininosuccinic aciduria
- Citrullinemia
- Phenylketonuria
- Hyperargininemia ✔
- Elevated blood argininosuccinate level is found in
- Hyperargininemia
- Argininosuccinic aciduria ✔
- Citrullinemia
- Tyrosinosis
- Hyperargininemia, a defect in urea synthesis develops due to deficiency of the enzyme:
- Ornithine transcarbamoylase
- Argininosuccinase
- Arginase ✔
- Argininosuccinate synthetase
The questions are typically designed to assess the technical skills and knowledge required for the laboratory profession, including the ability to analyze laboratory test results, perform laboratory procedures, and maintain laboratory equipment.
To prepare for these MCQs, candidates should have a thorough understanding of the key concepts and principles of laboratory science. They should also be familiar with common laboratory equipment and procedures, as well as laboratory safety protocols.
Candidates may also benefit from studying specific laboratory science textbooks or taking online courses that cover the material tested in the MCQs. Additionally, practicing sample MCQs and reviewing the answers can help candidates identify areas where they may need to improve their knowledge or skills.
Overall, the MCQs for lab technologists are designed to be challenging and comprehensive, requiring candidates to demonstrate a high level of proficiency in the field of laboratory science.
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